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Progressively shortening telomeres in mice model found to be responsible for the cardiomyopathy that is fatal in Duchenne muscular dystrophy. This is the first time that telomere shortening has been directly linked to mitochondrial function via a DNA damage response in non-dividing cells.
Progressively shortening telomeres in mice model found to be responsible for the cardiomyopathy that is fatal in Duchenne muscular dystrophy. This is the first time that telomere shortening has been directly linked to mitochondrial function via a DNA damage response in non-dividing cells.
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